Why does kidney failure have hyperphosphatemia?
Phosphorus is one of the most abundant elements in human body, second only to calcium.
The body's absorption of phosphorus is easier than calcium, so phosphorus deficiency is generally not present.
Why does kidney failure have hyperphosphatemia?
Under normal conditions, phosphorus is consumed through diet and the removal of phosphorus is mainly done by the kidneys.
The combination of phosphorus from the intestinal tract and calcium, which limits the absorption of calcium, and the hypoalbuminemia of kidney disease, as well as the formation barrier after kidney disease, can reduce the blood calcium.
High blood phosphorus and low blood calcium stimulate the parathyroid gland, which causes hyperthyroidism in secondary parathyroid, which leads to the bone calcification barrier.
How can hemodialysis patients effectively control the blood phosphorus?
1. Full dialysis can help control hyperphosphatemia, but the majority of phosphorus in the body is in human cells, so hemodialysis can remove only a small amount of phosphorus.
2, protein is the main food of phosphorus, the blood dialysis patients should receive high quality protein diet, restricted diet reduce phosphorus is taken while improve hyperphosphatemia, but low phosphorus diet to maintain adequate nutrition and adequate protein is taken is unfavorable, and malnutrition and protein were insufficient exists generally in dialysis patients.
3. Using the phosphorus binding agent, the phosphorus in the gastrointestinal tract and diet can be combined to form a material that cannot be absorbed and then excreted through the excrement, thus reducing the absorption of phosphorus in the intestinal tract.
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